Please use this identifier to cite or link to this item: http://hdl.handle.net/11455/69377
標題: Foodborne disease outbreaks caused by sucrose-nonfermenting and beta-galactosidase-deficient variants of Vibrio cholerae
作者: Wei, S.W.
Chern, L.L.
Wu, Y.C.
Wang, Y.L.
Lin, C.M.
Chiou, C.S.
關鍵字: foodborne disease outbreak
Vibrio cholerae
molecular identification
polymerase-chain-reaction
molecular-identification
family
vibrionaceae
hemolysin
bacteria
sequence
strains
non-o1
taiwan
o139
期刊/報告no:: International Journal of Food Microbiology, Volume 122, Issue 1-2, Page(s) 148-155.
摘要: We reported four foodborne disease outbreaks in Taiwan caused by sucrose-nonfermenting and by beta-galactosidase-deficient variants of non-O1, non-O139 Vibrio cholerae. The sucrose-nonfermenting vibrios collected from three outbreaks were biochemically identified to be V mimicus and the beta-galactosidase-deficient vibrios from an Outbreak to be V alginolyticus. However, molecular methods including DNA-DNA hybridization, fatty acid profile analysis, and sequence analysis of 16S rRNA, oriC, pyrH, recA, and rpoA indicated that these vibrios should be V cholerae. These V cholerae variants carried two hemolysin genes, hlyA and hlx, but contained neither cholera toxin gene, ctx, V mimicus hemolysin gene, vmh, nor then-no-directed hemolysin, tdh. The sucrose-nonfermenting variants of V cholerae shared a high level of genetic relatedness; they could derive from a common clone. In our record from 1995 to date, this was the first time that V cholerae variants were discovered as etiologic agents for foodborne disease outbreaks in Taiwan. (c) 2007 Elsevier B.V. All rights reserved.
URI: http://hdl.handle.net/11455/69377
ISSN: 0168-1605
文章連結: http://dx.doi.org/10.1016/j.ijfoodmicro.2007.11.061
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