Please use this identifier to cite or link to this item: http://hdl.handle.net/11455/94017
DC FieldValueLanguage
dc.contributor.authorWang, Sin-Tingzh_TW
dc.contributor.authorHuang, Shi-Weizh_TW
dc.contributor.authorKao, Jun-Kaizh_TW
dc.contributor.authorLiang, Shu-Meizh_TW
dc.contributor.authorChen, Yi-Juzh_TW
dc.contributor.authorChen, Yu-Yuzh_TW
dc.contributor.authorWu, Chun-Yingzh_TW
dc.contributor.authorShieh, Jeng-Jerzh_TW
dc.date2015-05-
dc.date.accessioned2016-08-08T06:55:16Z-
dc.date.available2016-08-08T06:55:16Z-
dc.identifier.urihttp://hdl.handle.net/11455/94017-
dc.description.abstractAMP-activated protein kinase (AMPK), a principal intracellular energy sensor, plays a crucial role in cell growth, proliferation, apoptosis and autophagy. Imiquimod (IMQ) directly exhibits anti-tumor activity through the induction of apoptosis and autophagic cell death.zh_TW
dc.language.isoenzh_TW
dc.relationJournal of dermatological science, Volume 78, Issue 2, Page(s) 108-16.zh_TW
dc.subjectAMPKzh_TW
dc.subjectApoptosiszh_TW
dc.subjectAutophagyzh_TW
dc.subjectImiquimodzh_TW
dc.subjectAMP-Activated Protein Kinaseszh_TW
dc.subjectAdenosine Triphosphatezh_TW
dc.subjectAminoquinolineszh_TW
dc.subjectAntineoplastic Agentszh_TW
dc.subjectApoptosiszh_TW
dc.subjectAutophagyzh_TW
dc.subjectCarcinoma, Basal Cellzh_TW
dc.subjectCarcinoma, Squamous Cellzh_TW
dc.subjectCell Line, Tumorzh_TW
dc.subjectDown-Regulationzh_TW
dc.subjectEnzyme Activationzh_TW
dc.subjectGene Silencingzh_TW
dc.subjectHumanszh_TW
dc.subjectMelanomazh_TW
dc.subjectMyeloid Cell Leukemia Sequence 1 Proteinzh_TW
dc.subjectMyeloid Differentiation Factor 88zh_TW
dc.subjectPhosphorylationzh_TW
dc.subjectProtein Biosynthesiszh_TW
dc.subjectProtein-Serine-Threonine Kinaseszh_TW
dc.subjectSignal Transductionzh_TW
dc.subjectSkin Neoplasmszh_TW
dc.subjectToll-Like Receptor 7zh_TW
dc.subjectToll-Like Receptor 8zh_TW
dc.titleImiquimod-induced AMPK activation causes translation attenuation and apoptosis but not autophagyzh_TW
dc.typeJournal Articlezh_TW
dc.identifier.doi10.1016/j.jdermsci.2015.02.008zh_TW
item.languageiso639-1en-
item.fulltextno fulltext-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.grantfulltextnone-
item.cerifentitytypePublications-
item.openairetypeJournal Article-
Appears in Collections:生物醫學研究所
Show simple item record
 

Google ScholarTM

Check

Altmetric

Altmetric


Items in DSpace are protected by copyright, with all rights reserved, unless otherwise indicated.